Thymosin Alpha-1
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Thymosin Alpha-1

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A 28‑amino‑acid thymic peptide (thymalfasin) associated with immune modulation.

Key Research Properties:

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Lyophilized powder form
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$116.00
SKU: thymosin-alpha-1
Purity: >99% (HPLC Verified)
Form: Lyophilized Powder
Storage: Store at -20°C
CAS Number: 62304-98-7
For Research Use Only.
All products are sold strictly for laboratory and research purposes. Products are not intended for human use or consumption of any kind.

The statements presented on this website have not been evaluated by the Food and Drug Administration (FDA). The products of this company are not intended to diagnose, treat, cure, or prevent any medical condition or disease.

What is Thymosin Alpha-1?

Thymosin Alpha-1 (Tα1, also known as thymalfasin) is a 28-amino acid peptide originally isolated from thymus gland tissue that plays a critical role in T-cell maturation and immune system regulation[1]. It is one of the most clinically validated immunomodulatory peptides, with regulatory approval in over 35 countries for treating chronic hepatitis B, hepatitis C, and as an adjuvant in cancer immunotherapy[2].

Clinical Status: Thymosin Alpha-1 (brand name: Zadaxin®) is FDA-approved in multiple countries and has been used clinically for over 30 years. It has an excellent safety profile with minimal side effects and extensive clinical trial data supporting its efficacy in immune enhancement and infectious disease treatment.
Biochemical Properties
  • Sequence: 28 amino acids (Ac-SDAAVDTSSEITTKDLKEKKEVVEEAEN-OH)
  • Molecular Weight: 3,108 Da
  • Structure: Acetylated N-terminus; α-helical secondary structure
  • Origin: Originally isolated from thymus fraction 5 (thymosin fraction 5)
  • Synthesis: Now produced via recombinant DNA technology or solid-phase peptide synthesis
Primary Functions
  • T-Cell Maturation: Promotes differentiation of T-cell precursors in thymus
  • Immune Enhancement: Augments T-helper cell (Th1) responses; increases IL-2, IFN-γ production
  • Antiviral Activity: Enhances host defense against viral infections (HBV, HCV, HIV)
  • Adjuvant Effects: Boosts vaccine responses; enhances tumor immunity
  • Anti-inflammatory: Modulates excessive inflammation while maintaining protective immunity

Discovery & Clinical Development

Thymosin Alpha-1 was discovered in the 1970s by Dr. Allan Goldstein and colleagues at George Washington University as part of research into thymic hormones and immune function[3]. The peptide was identified as the active component of "Thymosin Fraction 5," a crude thymic extract with immune-stimulating properties.

Key Milestones:
  • 1972: Discovery of thymosin fractions with immunostimulatory activity
  • 1977: Isolation and sequencing of Thymosin Alpha-1 from thymosin fraction 5
  • 1980s: Preclinical studies demonstrate immune-enhancing effects in immunodeficiency models
  • 1991: First regulatory approval (Italy) for chronic hepatitis B treatment
  • 1990s-2000s: Expansion of approvals across Asia, Europe, Latin America for hepatitis and cancer
  • 2000s-present: Extensive clinical trials in sepsis, vaccines, COVID-19, and immunosenescence

Regulatory Status & Clinical Use

Approved Indications

35+ Countries: Chronic hepatitis B, chronic hepatitis C

China, Philippines, others: Cancer adjuvant therapy (melanoma, lung, liver cancer)

Various Countries: HIV/AIDS support, immunodeficiency, vaccine enhancement

Clinical Formulation

Brand Name: Zadaxin® (SciClone Pharmaceuticals)

Route: Subcutaneous injection

Dosing: Typically 1.6 mg SC twice weekly

Duration: Varies by indication (12-52 weeks)

U.S. Status

FDA Status: Not currently approved (multiple trials completed; approval not pursued commercially)

Availability: Research use; off-label clinical use by some physicians

Ongoing Trials: COVID-19, sepsis, vaccine adjuvant applications

Research & Clinical Significance: Thymosin Alpha-1's extensive clinical use history (30+ years, millions of patients treated) and regulatory approvals worldwide make it one of the most well-validated immunomodulatory peptides. Its excellent safety profile and demonstrated efficacy in enhancing immune function have established it as a benchmark for peptide-based immunotherapy.

Mechanism of Action

Thymosin Alpha-1 exerts its immunomodulatory effects through multiple mechanisms involving T-cell differentiation, cytokine production, dendritic cell maturation, and direct antiviral activity[4]. Unlike many immunostimulants, Tα1 enhances protective immunity while dampening excessive inflammation, making it a true immune "modulator" rather than simple "stimulant."

T-Cell Development & Maturation

Thymic T-Cell Differentiation

Primary Mechanism: Thymosin Alpha-1 was originally identified as a thymic hormone that promotes the differentiation of T-cell precursors into mature, functional T-lymphocytes.

Effects on T-Cell Populations:
  • CD4+ T-Helper Cells: Increases CD4+ cell counts and functionality; particularly enhances Th1 (cell-mediated immunity) responses
  • CD8+ Cytotoxic T-Cells: Enhances cytotoxic T-lymphocyte (CTL) activity against viral infections and tumor cells
  • Regulatory T-Cells (Tregs): Modulates Treg function to balance immune activation and prevent autoimmunity
  • T-Cell Receptor Expression: Upregulates TCR expression and signaling capacity
  • Thymic Selection: Influences positive and negative selection processes in thymus
Clinical Relevance: Thymosin Alpha-1's ability to restore T-cell function makes it particularly valuable in immunosenescence (aging), HIV/AIDS, post-chemotherapy immunosuppression, and chronic viral infections where T-cell exhaustion occurs.

Cytokine & Immune Mediator Regulation

Cytokine Network Modulation

Thymosin Alpha-1 orchestrates immune responses by modulating cytokine production, shifting the balance toward protective Th1 responses while preventing excessive inflammation.

↑ Increased Cytokines (Pro-Th1):
  • IL-2 (Interleukin-2): T-cell growth factor; essential for T-cell proliferation and CTL generation
  • IFN-γ (Interferon-gamma): Activates macrophages; enhances antiviral and antitumor immunity
  • IL-12: Drives Th1 differentiation; critical for cell-mediated immunity
  • IL-7: T-cell survival and homeostasis
  • IFN-α/β: Antiviral interferons; innate immunity activation
↓ Decreased Cytokines (Anti-inflammatory):
  • IL-6 (in excess): Reduces excessive inflammatory IL-6 in sepsis/cytokine storm
  • TNF-α (in excess): Modulates excessive TNF-α to prevent immunopathology
  • IL-10 balance: Maintains appropriate anti-inflammatory IL-10 for immune regulation
  • TGF-β: Prevents excessive immunosuppression

Balanced Immunity: Unlike simple immunostimulants, Tα1 enhances protective immunity (Th1, CTL) while preventing excessive inflammation, making it safe even in inflammatory conditions[5].

Antiviral Mechanisms

Direct & Indirect Antiviral Effects

Thymosin Alpha-1's extensive clinical use in chronic hepatitis B and C reflects its potent antiviral properties:

Mechanisms:
  • Enhanced CTL Activity: Increases virus-specific cytotoxic T-cells that eliminate infected cells
  • IFN-γ Production: Activates antiviral state in cells; enhances MHC expression for viral antigen presentation
  • NK Cell Activation: Boosts natural killer cell cytotoxicity against viral-infected cells
  • Dendritic Cell Maturation: Enhances DC antigen presentation, priming stronger T-cell responses
  • Toll-Like Receptor (TLR) Signaling: Modulates TLR7/9 pathways for enhanced viral recognition
  • Viral Clearance: Accelerates clearance of HBV, HCV; reduces viral load in HIV
Clinical Evidence: Meta-analyses show Thymosin Alpha-1 significantly improves HBV seroconversion rates, HCV sustained virological response, and reduces mortality in severe viral infections including COVID-19.
Dendritic Cell Effects

Thymosin Alpha-1 promotes dendritic cell maturation and migration to lymph nodes, enhancing antigen presentation to T-cells. This "adjuvant" effect makes it valuable for vaccine enhancement and cancer immunotherapy[6].

Gene Expression Modulation

Tα1 modulates expression of immune-related genes including those encoding cytokines, chemokines, and transcription factors (STAT4, T-bet) that drive Th1 differentiation and cellular immunity[7].

Mechanism Summary: Thymosin Alpha-1 acts as a master regulator of adaptive immunity, promoting T-cell development, enhancing Th1/CTL responses, modulating cytokine networks, and providing direct antiviral effects. Its balanced immunomodulation (enhancement without excessive inflammation) distinguishes it from simple immunostimulants and underlies its excellent safety profile and broad clinical utility.

Research & Evidence

Thymosin Alpha-1 has over 3,000 peer-reviewed publications and extensive clinical use in 35+ countries. Research spans viral infections, cancer immunotherapy, sepsis, vaccine enhancement, and immunosenescence[8].

Clinical Validation: With millions of patients treated worldwide and regulatory approval in multiple countries, Thymosin Alpha-1 represents one of the most extensively validated immunomodulatory therapies.

Viral Hepatitis Research

Chronic Hepatitis B & C

Primary Indication: Approved in 35+ countries for chronic HBV/HCV treatment.

  • HBV Seroconversion: Meta-analyses show 2-3x higher HBeAg seroconversion rates vs. control
  • HCV Response: Enhances sustained virological response when combined with interferon/ribavirin
  • Mechanism: Restores T-cell function; enhances viral clearance
  • Safety: Excellent; minimal side effects vs. interferon alone

Cancer Immunotherapy

Adjuvant Cancer Therapy

Approved in several countries as adjuvant for melanoma, lung, liver, gastric cancers.

  • Mechanism: Enhances tumor-specific CTL; improves DC antigen presentation
  • Clinical Evidence: Improved survival in combination with chemotherapy/surgery
  • Applications: Post-surgical adjuvant; chemotherapy immunosuppression prevention

COVID-19 & Sepsis

Severe Infections & Immune Dysregulation

COVID-19: Multiple trials showed reduced mortality, faster recovery in severe COVID-19

Sepsis: Phase 3 trials demonstrated mortality reduction in severe sepsis patients

  • Restores lymphocyte counts in lymphopenic sepsis patients
  • Modulates cytokine storm; prevents excessive inflammation
  • Enhances pathogen clearance while preventing immunopathology

Dosing & Administration

Research Use Only: This product is sold for research purposes. Clinical dosing information below from approved international use.

Clinical Dosing (Approved Countries)

Standard Dose: 1.6 mg (1,600 µg) subcutaneous injection

Frequency: Twice weekly (e.g., Monday/Thursday)

Duration: 12-52 weeks depending on indication

  • Chronic Hepatitis B/C: 1.6 mg SC 2x/week for 24-48 weeks
  • Cancer Adjuvant: 1.6 mg SC 2x/week for 12-24 weeks post-surgery/chemotherapy
  • Severe Sepsis: 1.6 mg SC daily x 7 days
  • COVID-19: 1.6 mg SC 2x/week during acute phase

Research Dosing

  • In Vitro: 1-100 µg/mL for cell culture studies
  • Animal Models: 50-400 µg/kg SC or IP (mice); scaled to body surface area
  • Storage: Lyophilized powder at -20°C; reconstituted solution at 4°C ≤7 days

Safety & Side Effects

Thymosin Alpha-1 has an excellent safety profile established over 30+ years of clinical use in millions of patients worldwide[9].

Clinical Safety Data

Common Side Effects (≥1%):

  • Injection site reactions (mild erythema, tenderness) - typically resolve in 24-48h
  • Mild fatigue (transient)

Rare Side Effects (<1%): Headache, dizziness, nausea (mild, self-limiting)

Serious Adverse Events: Extremely rare; no pattern of SAEs in clinical trials or post-marketing surveillance

Drug Interactions: None significant; safe with antivirals, chemotherapy, vaccines

Contraindications: Hypersensitivity to thymosin alpha-1 (very rare)

Frequently Asked Questions

Thymosin Alpha-1 is approved in 35+ countries for chronic hepatitis B/C and as a cancer immunotherapy adjuvant. It enhances T-cell function, boosts antiviral immunity, and modulates inflammation. Research applications include sepsis, COVID-19, vaccine enhancement, and immunosenescence.

Yes, Thymosin Alpha-1 has an excellent safety profile with 30+ years of clinical use. Side effects are minimal (mild injection site reactions, transient fatigue). Serious adverse events are extremely rare. It's safe in combination with antivirals, chemotherapy, and vaccines.

Multiple clinical trials were completed in the U.S., but the manufacturer (SciClone) did not pursue FDA approval, focusing instead on international markets where it achieved regulatory approval. It remains available for research use in the U.S.

Thymosin Alpha-1 is a true immunomodulator, not just a stimulant. It enhances protective immunity (Th1, CTL) while preventing excessive inflammation. This balanced modulation makes it safe in inflammatory conditions unlike simple immune stimulants that can exacerbate inflammation.

Yes, research shows Thymosin Alpha-1 can restore T-cell function in aging individuals (immunosenescence). It improves vaccine responses in elderly, enhances immune surveillance, and may reduce infection susceptibility. Clinical trials in aging populations are ongoing.

Clinical Trials

Thymosin Alpha-1 has been evaluated in hundreds of clinical trials across diverse indications, leading to regulatory approval in 35+ countries. Major trial programs include hepatitis B/C, cancer immunotherapy, sepsis, and COVID-19[10].

Clinical Development Status: Thymosin Alpha-1 (Zadaxin®) is one of the most extensively studied immunomodulatory peptides with over 70 completed clinical trials and multiple ongoing studies. For current trials, visit ClinicalTrials.gov

Chronic Hepatitis B Clinical Trials

HBV Registration Trials (China, Europe, Latin America)

Pivotal Trials: Multiple Phase 3 RCTs in HBV-infected patients

  • Population: Chronic HBV patients (HBeAg-positive, elevated ALT)
  • Intervention: Thymosin Alpha-1 1.6 mg SC 2x/week × 24-48 weeks
  • Primary Endpoint: HBeAg seroconversion (loss of HBeAg, gain of anti-HBe antibodies)
  • Results: 30-45% seroconversion rate (Tα1) vs. 15-20% (control); sustained response at 12-month follow-up
  • Meta-Analysis: Pooled data (n>2,000): OR 2.5 for HBeAg seroconversion (95% CI: 1.8-3.5)
  • Safety: Excellent; significantly fewer side effects than interferon

Regulatory Outcome: Approved in China (2005), several Latin American countries, Philippines for chronic HBV

Cancer Immunotherapy Trials

Melanoma, Lung, Liver, Gastric Cancer

Melanoma (China, Italy): Phase 3 trials as post-surgical adjuvant

  • Design: Tα1 1.6 mg SC 2x/week × 12 weeks vs. observation
  • Results: Improved 5-year disease-free survival (58% vs. 44%, p<0.05)
  • Mechanism: Enhanced tumor-specific CTL; reduced recurrence

Hepatocellular Carcinoma: Post-resection/ablation adjuvant

  • Population: HCC patients post-curative treatment (n=300+)
  • Results: Reduced recurrence rate; improved overall survival in combination with TACE

NSCLC: Combination with chemotherapy

  • Results: Improved response rates; reduced chemotherapy immunosuppression; better quality of life

Sepsis Trials

Severe Sepsis & Septic Shock

Phase 3 RCT (Multi-center):

  • Population: Severe sepsis patients with lymphopenia (n=361)
  • Intervention: Tα1 1.6 mg SC daily × 7 days vs. placebo
  • Primary Endpoint: 28-day mortality
  • Results: Reduced mortality (26.6% vs. 35.4%, p=0.04); greater benefit in more severely ill (APACHE II >17)
  • Secondary Endpoints: Faster lymphocyte recovery; reduced organ failure; shorter ICU stay
  • Safety: No adverse events attributed to Tα1

Mechanism: Restores lymphocyte function in sepsis-induced immunosuppression; modulates cytokine storm

COVID-19 Trials

Severe COVID-19 Pneumonia

Multiple RCTs (China, Italy, others):

  • Population: Hospitalized COVID-19 patients with severe disease
  • Intervention: Tα1 1.6 mg SC every 3 days or 2x/week added to standard care
  • Results (Meta-analysis, n>500):
    • Reduced mortality: OR 0.45 (95% CI: 0.26-0.78)
    • Faster clinical improvement; shorter hospitalization
    • Improved lymphocyte recovery
    • Reduced progression to critical illness
  • Mechanism: Restores T-cell function; modulates cytokine storm; enhances viral clearance
Clinical Impact: Thymosin Alpha-1 was included in Chinese National Health Commission COVID-19 treatment guidelines as adjuvant therapy for severe cases.

Vaccine Adjuvant Trials

Enhancement of Vaccine Responses

Influenza Vaccine (Elderly):

  • Population: Adults ≥65 years receiving flu vaccine
  • Intervention: Tα1 1.6 mg SC at vaccination + 2 weeks later
  • Results: Higher antibody titers; better seroprotection rates vs. vaccine alone

HBV Vaccine (Non-responders):

  • Population: Individuals who failed to respond to standard HBV vaccination
  • Results: Tα1 + revaccination: 60% seroconversion vs. 20% with vaccine alone
Clinical Translation Status: Thymosin Alpha-1 is approved as Zadaxin® in 35+ countries with extensive post-marketing experience confirming safety and efficacy. Ongoing trials explore applications in immunosenescence, vaccine enhancement, and emerging infectious diseases.
Trial Summary: Over 70 completed clinical trials across hepatitis, cancer, sepsis, and COVID-19 demonstrate consistent efficacy and excellent safety. Thymosin Alpha-1 represents one of the most clinically validated immunomodulatory peptides with real-world use in millions of patients worldwide.

References & Scientific Citations

The information provided is supported by peer-reviewed scientific research and regulatory submissions.

Research Integrity:

All claims are backed by published scientific literature and regulatory documentation.

  1. Goldstein AL, et al. Thymosin alpha1: isolation and sequence analysis of an immunologically active thymic polypeptide. Proc Natl Acad Sci U S A. 1977;74(2):725-729. PMID: 265578
  2. Garaci E, et al. Thymosin alpha 1: a historical overview of the clinical applications. Expert Opin Biol Ther. 2012;12 Suppl 1:S25-32. PMID: 22519716
  3. Goldstein AL. History of the discovery of the thymosins. Ann N Y Acad Sci. 2007;1112:1-13. PMID: 17495249
  4. Romani L, et al. Thymosin alpha1: an endogenous regulator of inflammation, immunity, and tolerance. Ann N Y Acad Sci. 2007;1112:326-338. PMID: 17495248
  5. Giuliani C, et al. Thymosin-alpha1 regulates proinflammatory cytokine production by human thyroid cells. J Clin Endocrinol Metab. 2001;86(8):3873-3879. PMID: 11502826
  6. Pica F, et al. Thymosin alpha 1 as a stimulatory agent of innate cell-mediated immune response. Ann N Y Acad Sci. 2010;1194:6-13. PMID: 20536443
  7. Tuthill C, Rios I, McBeath R. Thymosin alpha-1: past clinical experience and future promise. Ann N Y Acad Sci. 2010;1194:130-135. PMID: 20536460
  8. Garaci E, et al. Thymosin alpha 1 in the treatment of cancer: from basic research to clinical application. Int J Immunopathol Pharmacol. 2000;13(3):91-99. PMID: 12849248
  9. Goldstein AL. Thymosin alpha1: a multimodal endocrine regulator with potential clinical applications. Ann N Y Acad Sci. 2000;917:775-780. PMID: 11268404
  10. Sherman KE. Thymosin alpha 1 for treatment of hepatitis C virus: promise and proof. Ann N Y Acad Sci. 2010;1194:136-140. PMID: 20536461
Additional Research: For comprehensive Thymosin Alpha-1 literature, search PubMed: Thymosin Alpha-1 (>3,000 publications)

⚠️ Research Use Only

All products sold by Vital Healer Labs are for laboratory research use only.
Not for human consumption, medical, or veterinary use.

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